Part 1 | Diagnostic, treatment & rehabilitation strategies for patients with CNS Lyme disease

0.5 CME. This course describes typical neuro-Lyme clinical presentations and discusses the mechanisms of nerve injury that are caused by Lyme disease bacteria. 

Instructor

Nev Zubcevik, DO
Chief Medical Officer
Invisible International

Description

This course describes neuro-Lyme clinical presentations and discusses the foundational treatment strategies used to get patients back to living normal lives. Based on Dr. Zubcevik’s experience as a Harvard-trained, board-certified physical medicine and rehabilitation physician, she stresses the importance of a multidisciplinary “all hands on deck” approach for these patients, many of whom have serious deficits in memory and brain functioning. She recommends that coordination of care —appointment management, home support, physician referrals, and insurance coverage—be an integral part of any treatment plan. She says that mental health support and an anti-inflammatory diet are also key to a patient’s recovery. This is part 1 in a three course series on this topic.

Learning objectives

  1. Define Central Nervous System Lyme (CNS) Disease
  2. Describe ways to initialize differential diagnosis and diagnostic tools
  3. Identify three ways of approaching rehabilitation strategies for CNS Lyme

This session, Part 1 | Diagnostic, treatment & rehabilitation strategies for patients with CNS Lyme disease, is approved for 0.5 enduring AAFP Prescribed credits.

AAFP Prescribed credit is accepted by the American Medical Association as equivalent to AMA PRA Category 1 credit(s)™ toward the AMA Physician’s Recognition Award. When applying for the AMA PRA, Prescribed credit earned must be reported as Prescribed, not as Category 1.

The AAFP has reviewed One Health Medical Education for a Changing Climate and deemed it acceptable for AAFP credit. Term of approval is from 01/02/2024 to 01/01/2025. Physicians should claim only the credit commensurate with the extent of their participation in the activity.

References

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Allen C. Steere, Gail McHugh, Nitin Damle, Vijay K. Sikand, Prospective Study of Serologic Tests for Lyme Disease, Clinical Infectious Diseases, Volume 47, Issue 2, 15 July 2008, Pages 188–195, https://doi.org/10.1086/589242

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Miklossy, J., Kasas, S., Zurn, A.D. et al. Persisting atypical and cystic forms of Borrelia burgdorferiand local inflammation in Lyme neuroborreliosis. J Neuroinflammation 5, 40 (2008). https://doi.org/10.1186/1742-2094-5-40

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Zubcevik N, Mao C, Wang QM, Bose EL, Octavien RN, Crandell D, Wood LJ. Symptom Clusters and Functional Impairment in Individuals Treated for Lyme Borreliosis. Front Med (Lausanne). 2020 Aug 21;7:464. doi: 10.3389/fmed.2020.00464. PMID: 32974369; PMCID: PMC7472530.15) Belman AL, Iyer M, Coyle PK, Dattwyler R. Neurologic manifestations in children with North American Lyme disease. Neurology. 1993 Dec;43(12):2609-14. doi: 10.1212/wnl.43.12.2609. PMID: 8255465.16) 

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Fallon BA, Zubcevik N, Bennett C, Doshi S, Rebman AW, Kishon R, Moeller JR, Octavien NR, Aucott JN. The General Symptom Questionnaire-30 (GSQ-30): A Brief Measure of Multi-System Symptom Burden in Lyme Disease. Front Med (Lausanne). 2019 Dec 6;6:283. doi: 10.3389/fmed.2019.00283. PMID: 31867334; PMCID: PMC6908481.

Nucl Med Commun. 2002 Aug;23(8):773-7.Cerebral metabolic changes associated with Lyme disease.Newberg A1, Hassan A, Alavi A.

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About Instructor

Not Enrolled

Course Includes

  • 1 Lesson
  • 2 Quizzes
  • Course Certificate